Sample report · Example data

Nutritional counselling · Practice and clinic

Metabolic Health

What your client eats, how they process it — and what they can change.

Parameters: 4 epigenetic + 17 genetic
Molecular level: Epigenetics and genetics
For tracking over time: the 4 epigenetic markers only
Genetic analysis: yes
Result format: Traffic light 1–9

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At a glance

Every value in this report

Each value with its classification. One click jumps to the full explanation below.

Sample report · Example data

8 outside the range9 borderline9 within range

LINE-1 · DNA stability2 / 9Outside range
Inflammation regulation (IL-6 & TNF-α)7 / 9Optimal
miR-21 · Inflammation & micronutrients3 / 9Outside range
miR-155 · FTO regulation, adipose tissue8 / 9Optimal
Skin hydration9 / 9Favourable
Difficulty maintaining weight8 / 9Favourable
Salt sensitivity8 / 9Favourable
Body weight regulation7 / 9Favourable
Vitamin D7 / 9Favourable
Oxidative cell protection7 / 9Favourable
Food cravings6 / 9Moderate
Skin elasticity & inflammation6 / 9Moderate
Difficulty losing weight5 / 9Moderate
B vitamins5 / 9Moderate
Hunger/satiety regulation4 / 9Moderate
Antioxidant capacity4 / 9Moderate
Blood sugar and insulin metabolism3 / 9Increased need
Blood lipids & fatty acid metabolism2 / 9Increased need
Omega-3 supply2 / 9Increased need
Folate2 / 9Increased need
Weight loss through exercise2 / 9Increased need
Hunger and satiety regulation4 / 9Moderate
Losing weight5 / 9
Maintaining weight8 / 9
Blood lipids and fatty acid metabolism2 / 9Increased need
Skin elasticity and inflammation6 / 9Moderate

The principle

How to read this report.

This report combines two levels: your genetics — inherited and unchangeable — and your epigenetics, which can be influenced by lifestyle.

Level 1 · Genetics

Your genes are the blueprint of your metabolism: how you process carbohydrates, fats and proteins, how appetite and satiety work, how your body handles vitamins and salt. These predispositions do not change.

SNP — a well-researched variant in your DNA.
Three forms — wild type, heterozygous or homozygous; without any value judgement.
Your profile — several SNPs together produce your metabolic type and evaluations.

Level 2 · Epigenetics

Epigenetics does not show which genes you have, but how active they currently are. These four values can be changed — and are therefore the only ones in which the effect of an intervention can be demonstrated over time.

LINE-1 — global DNA regulation and stability.
Inflammation regulation — combined score from IL-6 and TNF-α.
miR-21 · miR-155 — microRNAs of metabolic activity.

Orientation

How do I read the scale?

All evaluations — genetic and epigenetic alike — use the same traffic light 1–9. Red on the left, green on the right: 1–3 red, 4–6 amber, 7–9 green. Higher is always more favourable.

13579

1–3 · Red: Outside the optimal range. This is where the greatest leverage lies.
4–6 · Amber: Moderate need for support. Monitor specifically.
7–9 · Green: In the favourable range. Maintain existing routines.

The miniature

The report in miniature.

This miniature shows in advance which result formats this report uses. It is calculated from the sample values on this page and is not a picture. Each mark stands for a format that reappears at full size further down — and whatever this panel does not use is missing here as well.

1
The traffic light 1–9
Every evaluation sits as a mark on the same traffic light 1–9. Red on the left, green on the right, higher is more favourable. How the marks spread across the band shows directly whether this panel sits more towards the red, amber or green range.

Important notices

Sample report: All values, genotypes and personal details in this document are fictitious and serve solely to illustrate the report format. This is not a real report.
Interpretation: This report is not a diagnosis and does not replace medical advice. Genetic evaluations describe predispositions, not diseases; epigenetic markers describe a momentary state. Both are to be assessed in the individual clinical context by qualified professionals.
Status of the analyses: The analyses are laboratory services provided by our cooperation partner and are not CE-marked in vitro diagnostic devices. They serve consultation and monitoring and do not replace clinical diagnostics. The analyses are supplied to professional users only.
Legal framework: Genetic testing is subject to the German Genetic Diagnostics Act (GenDG); information and written consent are required before sample collection. The laboratory analysis is carried out by our cooperation partner. Polaris Diagnostics Europe GmbH is responsible for distribution and support. This document does not constitute any promise of cure within the meaning of the German Therapeutic Products Advertising Act (Heilmittelwerbegesetz).

Important

This report is not a diagnosis. Interpretation takes place in consultation with qualified professionals and always in the individual context.
Information and written consent are required before a genetic analysis (German Genetic Diagnostics Act, GenDG). Predictive genetic testing may only be conducted under the responsibility of appropriately qualified physicians; genetic counselling must be offered before and after the test (Sections 7 and 10 GenDG).
The genetic values never change. Only the four epigenetic values are suitable for monitoring over time.

How this becomes a consultation

A report is a programme, not a single value.

01
Before the consultation
The full report is available ahead of the appointment. Read it through and mark what stands out: the traffic-light ratings, their distribution across the evaluations, the starting points named. That way it is clear from the outset what to discuss.
02
During the consultation
Four values in this panel sit on the epigenetic level and can therefore be influenced. The genetic variants alongside them are determined once in a lifetime and remain unchanged; they describe the starting position. Only the epigenetic level responds to nutrition, exercise, sleep and strain — it alone is suitable for tracking progress.
03
Afterwards
A follow-up measurement is meaningful at the earliest after four to six months; before that, changes cannot be measured reliably. The interval is set by the supervising institution. For comparability, the laboratory, sample material and sampling should stay the same — and whatever happens between the measurements belongs in the same documentation as the values.

Sample evaluation

The values below come from a complete sample evaluation — exactly as your client receives it. Sample data, not a real person.

Report for: Muster, Erika
Sample collection: 08.04.2026
Material: Dried blood spot card
Evaluations: 17 + 4 epigenetic

Results at a glance

Your four modifiable values.

These four markers change through diet, exercise, sleep and stress — the reason for a follow-up measurement.

Marker	Level	Your value	Optimal	Interpretation
LINE-1 · DNA stability	Methylation	2 / 9	7–9	Outside range
Inflammation regulation (IL-6 & TNF-α)	Methylation	7 / 9	7–9	Optimal
miR-21 · Inflammation & micronutrients	microRNA	3 / 9	7–9	Outside range
miR-155 · FTO regulation, adipose tissue	microRNA	8 / 9	7–9	Optimal

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Your metabolic type: balanced type

Your metabolism does not favour any one of the three macronutrients. The distribution and calculated energy requirement follow in the “Metabolic type” chapter.

Your exercise type: endurance type

Longer, steady exertion suits you better than short bursts of strength — a strength component nevertheless remains important.

What follows from this

Two red epigenetic values — LINE-1 (2/9) and miR-21 (3/9); the genetic MTHFR variant (2/9) depends on the same metabolic pathway. This is the central starting point of this report.
Two green values — inflammation regulation (7/9) and miR-155 (8/9) are in the optimal range and should be kept there.
A follow-up measurement is meaningful after 4 to 6 months at the earliest.

All genetic evaluations

17 values

Results at a glance

Seventeen evaluations on the traffic light 1–9. These values are inherited and do not change — they describe where your metabolism needs more support than average.

Skin hydrationAQPR9/9· Favourable

Difficulty maintaining weight8/9· Favourable

Salt sensitivity8/9· Favourable

Body weight regulation7/9· Favourable

Vitamin DTransport, GC7/9· Favourable

Oxidative cell protectionSOD27/9· Favourable

Food cravings6/9· Moderate

Skin elasticity & inflammationIL-66/9· Moderate

Difficulty losing weight5/9· Moderate

B vitaminsRequirement5/9· Moderate

Hunger/satiety regulation4/9· Moderate

Antioxidant capacityNADPH4/9· Moderate

Blood sugar and insulin metabolism3/9· Increased need

Blood lipids & fatty acid metabolism2/9· Increased need

Omega-3 supplyALA → EPA/DHA2/9· Increased need

FolateMTHFR2/9· Increased need

Weight loss through exerciseFTO2/9· Increased need

Sorted by value, not by topic. The detailed interpretation with genotypes and interventions follows in the chapters below.

Methylation

2 values

Epigenetic status

DNA methylation shows how active your genes currently are. Unlike the genes themselves, these values can be changed.

LINE-1DNA stability

LINE-1 elements make up around 17 % of the genome. Their methylation is regarded as a marker of global DNA regulation.

2 / 9Outside range

159

This marker is most effectively supported through the nutrients involved in folate metabolism — which gains additional importance given your MTHFR variant (genetic 2/9).

Measure	Source	Amount / frequency	Effect
Folic acid	green leafy vegetables, tomato, pulses, nuts, sprouts	daily	If needed, 2 months as methylfolate (5-MTHF)
Riboflavin (B2)	germinated seeds, sprouts, cheese	daily	Cofactor of methylation
Cobalamin (B12)	egg, cheese, fermented foods	daily	Regeneration of active folate
Methionine	Brazil nuts, sesame, soya, peas, leafy vegetables	daily	Direct methyl group donor
Pyridoxine (B6)	cabbage, green beans, lentils, bananas	daily	Cofactor of methylation
Fibre	wholegrains, vegetables, pulses	at least 30 g/day	Bioavailability of phytochemicals

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Inflammation regulation (IL-6 & TNF-α)

Combined score from the methylation of both gene regions — not the acute blood level. Individual values: IL-6 8/9 · TNF-α 6/9.

7 / 9Optimal

159

A plant-focused, low-inflammatory diet continues to support this area.

Measure	Source	Amount / frequency	Effect
Resveratrol	grape skins, raspberry seeds, blueberries, peanuts	1 portion of berries/day	Found mainly in skins and seeds
Sulforaphane	broccoli, cauliflower, Brussels sprouts, cress	3–4×/week	Steam only briefly or eat raw — otherwise the active substance is lost
Quercetin	onions, garlic, capers, chives	daily	Let onions stand for 10 min after chopping
Curcumin	turmeric with black pepper	1 tsp daily	Oil improves absorption; not during pregnancy or with gallstones

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microRNA

2 values

Epigenetic status

microRNAs fine-tune the activity of your genes and reflect current metabolic and inflammatory activity. Like methylation, they can be influenced through lifestyle.

miR-21Inflammation and micronutrients

Worsens with more pronounced inflammatory activity as well as with a poor zinc and folate supply.

3 / 9Outside range

159

With a red or amber value, a normal BMI should be aimed for; in addition, targeted zinc and folate intake is worthwhile. Antioxidant micronutrients such as vitamin D, vitamin E and lycopene as well as resveratrol support immune regulation.

Measure	Source	Amount / frequency	Effect
Zinc	pumpkin seeds, oats, lentils, cheese, beef	daily	A poor zinc supply worsens this value
Folate	leafy vegetables, pulses, tomato, sprouts	daily	A poor folate supply worsens this value
Resveratrol	grape skins, blueberries, peanuts	1 portion of berries/day	Inhibits the NF-κB signalling pathway
Vitamin D	salmon, butter, egg, avocado, mushrooms	regularly	Important for immune regulation
Vitamin E	wheatgerm oil, nuts, spinach, broccoli	daily	Antioxidant, protects cell membranes
Lycopene	tomatoes, melon, carrots, papaya	daily	Carotenoid with an antioxidant effect

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miR-155FTO regulation and adipose tissue formation

Central regulator of immune and inflammatory responses; influences the formation of brown and beige adipose tissue.

8 / 9Optimal

159

Exercise, fasting and cold stimuli have a favourable effect here and influence the FTO effect positively — particularly relevant because your genetic FTO evaluation is 2/9.

Measure	Source	Amount / frequency	Effect
Regular exercise	endurance and strength combined	150 min/week	Has a favourable effect on this value
Cold stimuli	cold showers, ice bathing, cool rooms	regularly, briefly	Activates brown/beige adipose tissue
Fasting phases	extended overnight break from eating	after consultation	Supports the activation of brown adipose tissue
Quercetin	onions, garlic, capers, chives	at least 200 mg/day	Influences mitochondrial biogenesis

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Metabolic type: balanced type

3 values

Genetics · Macronutrients

Your metabolism does not particularly favour any one of the three macronutrients. The general recommendation of the DACH reference values therefore applies to you. More important in your case is the quality of the foods.

Share	Macronutrient	Note
55 %	Carbohydrates	Wholegrains, pulses and vegetables as the basis
20 %	Protein	20–30 g per main meal
25 %	Fat	Focus on unsaturated fats

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Individually calculated daily energy requirement: 1,900 kcal.

Exercise type: endurance type

2 values

Genetics · Exercise

Both ACE and ACTN3 point to a predisposition for endurance performance. For context: genetics explains only up to around 20 % of the difference between strength and endurance performance — training, diet and consistency have by far the greater influence.

Gene	Your genotype	Predisposition
ACE	GG (homozygous)	Rather endurance — variant associated with better oxygen utilisation and efficiency.
ACTN3	TT (wild type)	Rather endurance — XX type without α-actinin-3, more slow, endurance-oriented muscle fibres.

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Weight loss through exercise

Genetics · Exercise

Weight loss through exerciseFTO rs1121980

Genotype AT (heterozygous). How much exercise alone contributes to weight loss.

2 / 9Increased need

159

In your case, exercise alone leads to weight loss somewhat more slowly than with other genotypes. The effect on fitness and metabolic health remains unchanged — for weight reduction, the combination of exercise and dietary adjustment is particularly worthwhile.

Measure	Source	Amount / frequency	Effect
Endurance component	cycling, running, swimming	2–3×/week	Oxygen supply, basic endurance
Strength component	full-body strength training	2×/week	Maintains muscle mass and metabolic activity
Combine with diet	see metabolic type recommendation	individual	With FTO, the most effective additional lever

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Predisposition: body weight regulation and blood sugar metabolism

2 values

Genetics · Metabolism

Genetic predisposition for the regulation of body weight and for blood sugar metabolism — as a starting point for targeted nutrition, not as a prediction.

Body weight regulation

Summary of variants that are associated with the regulation of body weight.

7 / 9Genotypes: FTO rs9939609 TT · PPARG2 CC (wild type) · MC4R CT · TCF7L2 CT · SLC6A14 CA · TFAP2B GGFavourable

159

Your genetic background shows no unfavourable pattern here. Meal structure, fibre and regular exercise maintain this starting position.

Measure	Source	Amount / frequency	Effect
Fibre	wholegrains, pulses, vegetables	at least 30 g/day	Prolongs satiety, stabilises blood sugar
Protein per meal	eggs, pulses, fish, meat	20–30 g/meal	Supports satiety signals
Exercise	—	150 min/week	Attenuates genetically driven effects

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Blood sugar and insulin metabolism

Genes that influence insulin secretion, insulin sensitivity and blood sugar metabolism.

3 / 9Genotypes: TCF7L2 CT · IL-6 CG · ADRB3 GG (wild type) · PPARG2 CCIncreased need

159

Your value lies outside the favourable range. Carbohydrate quality and regular exercise are the most effective levers in your case — and, together with the red miR-21 value, the second focus of this report.

Measure	Source	Amount / frequency	Effect
Low glycaemic index	wholegrains, pulses, vegetables	every main meal	Avoids sharp blood sugar peaks
Movement after eating	short walk	10–15 min	Improves insulin sensitivity

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Nutrition type

4 values

Genetics · Eating behaviour

Satiety, food cravings and the tendency to lose or maintain weight — your genetic levers around eating behaviour.

Food cravings

6 / 9MC4R CT · FTO rs9939609 TT · LEPR AA · SLC6A14 CA · PPARG2 CCModerate

159

A slightly increased tendency towards spontaneous eating impulses. Meal structure and protein intake are your most effective levers here.

Measure	Source	Amount / frequency	Effect
Protein with every meal	eggs, pulses, fish, meat, tofu	20–30 g/meal	Strongest known satiety signal
Regular meals	—	3 fixed times/day	Reduces blood sugar fluctuations

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Hunger and satiety regulation

4 / 9MC4R, LEPR and FTO rs1121980 (AT) togetherModerate

159

A somewhat delayed satiety signal — slow, mindful eating gives this signal the time it needs.

Measure	Source	Amount / frequency	Effect
Slow, mindful eating	—	at least 20 min/meal	Gives the delayed signal time
High-fibre diet	vegetables, wholegrains	at least 30 g/day	Prolongs the feeling of fullness

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Losing weight

5 / 9ADRB3 GG · PLIN CT

159

Increased need for support with sleep and leptin sensitivity. 7–9 hrs of sleep and combined training 2–3×/week.

Maintaining weight

8 / 9TCF7L2 CT

159

Favourable starting position against the yo-yo effect. Stable meal times and 150 min of exercise/week maintain it.

Vitamin uptake

3 values

Genetics · Micronutrients

How well your body utilises vitamin D and converts folate into its active form — including the resulting requirement for B vitamins.

FolateMTHFR

Genotype homozygous variant. Key enzyme in folate metabolism.

2 / 9Increased need

159

Your MTHFR variant indicates a markedly limited conversion of folate into its active form (5-MTHF). Your requirement for folate from food is higher than with other genotypes. This value also explains the red LINE-1 marker on the epigenetic level — both depend on the same metabolic pathway.

Measure	Source	Amount / frequency	Effect
Folate from food	green leafy vegetables, pulses, asparagus	2–3 portions/day	MTHFR variants benefit from a consistently high intake
Active folate (5-MTHF)	if required, as a food supplement	after consultation with a doctor	Bypasses the limited conversion directly

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Vitamin DTransport

GC (vitamin D-binding protein), genotype GT (heterozygous)

7 / 9Favourable

159

Vitamin D transport in your blood is only slightly limited. This does not mean a deficiency, but it does make a blood level check worthwhile during the months with little sunlight.

Measure	Source	Amount / frequency	Effect
Vitamin D	oily fish, eggs; sunlight	15–20 min/day regularly, check levels	Checking is worthwhile above all in winter
Vitamin K2	fermented foods, egg yolk, hard cheese	daily	Supports vitamin D in bone and vascular metabolism

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B vitaminsRequirement

Follows from the MTHFR variant via the methylation cycle.

5 / 9Moderate

159

Measure	Source	Amount / frequency	Effect
Vitamin B2	eggs, dairy products, wholegrains	daily	Direct cofactor of the MTHFR enzyme
Vitamin B6	potatoes, pulses, bananas	daily	Homocysteine metabolism
Vitamin B12	eggs, fish, dairy products	daily	Regeneration of active folate

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Omega 3 and blood lipids

Genetics · Lipid metabolism

How well you utilise plant-based omega-3 precursors and how your blood lipid levels are genetically predisposed.

Blood lipids and fatty acid metabolism

2 / 9FADS1 TT · APOA5 AG · LPL AA (wild type) · PPARG2 CCIncreased need

159

Your FADS1 variant indicates that the body converts plant-based omega-3 precursors (ALA) into the active forms EPA and DHA only to a limited extent. The separate evaluation “Omega-3 supply” looks exclusively at this conversion step and is likewise 2 / 9.

Measure	Source	Amount / frequency	Effect
EPA and DHA directly	oily sea fish (salmon, mackerel, herring); alternatively algal oil	fish 2×/week or algal oil daily	Bypasses the limited FADS1 conversion
Plant-based omega-3 sources	linseed oil, walnuts, chia seeds	in addition, not as the sole source	In your case only partly converted
Limit omega-6	sunflower, safflower and corn oil sparingly	in everyday life	Favourable omega-6 to omega-3 ratio
Reduce sugar and white flour	in favour of wholegrains	in everyday life	Relates to triglyceride metabolism (APOA5)

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Salt sensitivity

Genetics · Blood pressure

Salt sensitivity

8 / 9ACE, genotype GG (homozygous)Favourable

159

Your ACE variant shows no increased salt sensitivity. As a general rule: anyone who eats a saltier diet should deliberately drink more.

Measure	Source	Amount / frequency	Effect
Keep an eye on salt intake	processed foods, ready meals	max. 6 g salt/day	Reduces water retention
Drink enough	water, unsweetened tea	at least 1.5–2 l/day	Supports sodium excretion
Potassium from vegetables and fruit	bananas, potatoes, green vegetables	daily	Counteracts salt-related retention

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Nutricosmetics

4 values

Genetics · Skin and antioxidants

Skin elasticity, hydration and antioxidant protection — how diet supports your skin from within.

Skin hydration

9 / 9AQPR CC (wild type)

159

Your skin retains moisture well — a normal care routine is usually sufficient. Supportive: at least 1.5–2 l of water daily.

Oxidative cell protection

7 / 9SOD2 AG (heterozygous)

159

Solid protection against oxidative stress. Daily antioxidants and consistent UV protection maintain this starting position.

Skin elasticity and inflammation

IL-6 CG (heterozygous). Increased activity can accelerate collagen breakdown.

6 / 9Moderate

159

A moderate tendency towards inflammation in the skin — an anti-inflammatory diet has a visible effect on your skin.

Measure	Source	Amount / frequency	Effect
Omega-3 fatty acids	salmon, linseed, walnuts	fish 2×/week	Supports the skin barrier
Antioxidants	berries, colourful vegetables, green tea	daily	Counteracts oxidative stress

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Antioxidant capacity

NADPH AG (heterozygous). Provides the reducing power for cellular antioxidant systems.

4 / 9Moderate

159

A moderately reduced antioxidant capacity — antioxidants through the diet provide additional support here.

Measure	Source	Amount / frequency	Effect
Antioxidants	berries, colourful vegetables, green tea	daily	Supports the cellular defence against free radicals
UV protection	sunscreen, headwear	when exposed to the sun	Limits UV exposure of the skin

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All genes analysed

21 values

Genetic basis

The complete list of the variants examined together with your genotype. These values are inherited and remain unchanged throughout life — repeating the genetic analysis serves no purpose.

Gene	Your genotype	Function
FTO rs9939609	TT (homozygous)	Risk allele A. Associated with BMI, body fat mass and waist circumference when the intake of saturated fatty acids is high.
FTO rs1121980	AT (heterozygous)	Risk allele A. Responds less well to weight loss through physical activity.
MTHFR	homozygous variant	Key enzyme in folate and methylation metabolism; influences the homocysteine level.
FADS1	TT (homozygous)	Controls the conversion of plant-based omega-3/-6 precursors into the active, long-chain fatty acids.
APOA5	AG (heterozygous)	Regulates the breakdown of triglycerides in the blood and influences blood lipid levels.
LPL	AA (wild type)	Breaks down fats from the blood; influences HDL cholesterol and blood lipid levels.
PPARG2	CC (wild type)	Regulates fat cell formation, fat storage and insulin sensitivity; influences appetite and satiety.
PLIN	CT (heterozygous)	Coats fat droplets in fat cells and regulates their build-up and breakdown.
ADRB3	GG (wild type)	Regulates fat burning in fat cells and the utilisation of stored fat.
MC4R	CT (heterozygous)	Centrally involved in controlling appetite, hunger and satiety signals in the brain.
LEPR	AA (homozygous)	Transmits the satiety signal of the hormone leptin.
SLC6A14	CA (heterozygous)	Transports amino acids; influences appetite, satiety and stress eating.
TCF7L2	CT (heterozygous)	Influences insulin secretion after carbohydrate-rich meals.
TFAP2B	GG (wild type)	Involved in the regulation of body weight and fat distribution.
IL-6	CG (heterozygous)	Central messenger substance of inflammation regulation; influences blood lipids, appetite and stress eating.
GC	GT (heterozygous)	Influences the transport of vitamin D in the blood.
ACE	GG (homozygous)	Part of the bradykinin-aldosterone system; influences endurance/strength predisposition and salt sensitivity.
ACTN3	TT (wild type)	Influences muscle fibre composition (fast vs. slow fibres).
AQPR	CC (wild type)	Influences water retention in the skin.
NADPH	AG (heterozygous)	Contributes to the antioxidant capacity of the cells.
SOD2	AG (heterozygous)	Important antioxidant enzyme, protects cells from oxidative stress.

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21 variants. Each of the evaluations in the chapter “All genetic evaluations” is derived from several of these genes together; a single genotype on its own does not allow any conclusion.

Summary

Your three focus areas.

From 21 genes and four epigenetic markers, three areas emerge in which effort and effect meet most clearly in your case.

Methylation metabolism — folate, B vitamins, zinc

Three values point in the same direction: LINE-1 2/9 (epigenetic), miR-21 3/9 (epigenetic) and folate/MTHFR 2/9 (genetic) — all three depend on the same metabolic pathway. This area is at the same time the only one whose effect can be demonstrated in a follow-up measurement. In concrete terms: green leafy vegetables and pulses daily, B2/B6/B12 via eggs, cheese and fermented foods, zinc via pumpkin seeds, oats and lentils. Active folate (5-MTHF) for two months after consultation with a doctor.

Fat quality — EPA and DHA directly

Blood lipids 2/9 and omega-3 supply 2/9. Your FADS1 variant makes plant-based omega-3 sources such as linseed oil and walnuts alone insufficient. Oily sea fish twice a week or algal oil daily bypasses this bottleneck directly; use omega-6-rich oils sparingly.

Blood sugar quality rather than calorie quantity

Blood sugar and insulin metabolism 3/9 alongside favourable body weight regulation (7/9). For you, the quality of the carbohydrates counts for more than the quantity: a low glycaemic index at every main meal, 20–30 g of protein per meal and a short walk after eating. Since exercise alone works more slowly in your case (FTO 2/9), this combination is the more effective route.

Follow-up and next step

A follow-up measurement of the four epigenetic markers is meaningful after 4 to 6 months at the earliest — before that, changes cannot be measured reliably.
The genetic evaluations remain unchanged and never need to be repeated.
A comparison of the measurement points over time is reported from the second measurement onwards.

Questions about your report?

Discuss the results with the facility that carried out the sample collection. That is where the clinical context needed for interpretation is available.

Contact

contact@polarisdx.net
+49 151 75011699
www.polarisdx.net

Distribution

Polaris Diagnostics Europe GmbH
Große Bleichen 1–3
20354 Hamburg

Sample report. All values, genotypes and personal details in this document are fictitious and serve solely to illustrate the report format. This is not a real report.

Interpretation. This report is not a diagnosis and does not replace medical advice. Genetic evaluations describe predispositions, not diseases; epigenetic markers describe a momentary state. Both are to be assessed in the individual clinical context by qualified professionals.

Status of the analyses. The analyses are laboratory services provided by our cooperation partner and are not CE-marked in vitro diagnostic devices. They serve consultation and monitoring and do not replace clinical diagnostics. The analyses are supplied to professional users only.

Legal framework. Genetic testing is subject to the German Genetic Diagnostics Act (GenDG); information and written consent are required before sample collection. The laboratory analysis is carried out by our cooperation partner. Polaris Diagnostics Europe GmbH is responsible for distribution and support. This document does not constitute any promise of cure within the meaning of the German Therapeutic Products Advertising Act (Heilmittelwerbegesetz).

All six documents are sample reports: every value, genotype and personal detail is fictitious and serves solely to illustrate the report format. These are not real results.

This report as a PDFRequest a quotation

Metabolic Health

Every value in this report

How to read this report.

Level 1 · Genetics

Level 2 · Epigenetics

How do I read the scale?

The report in miniature.

The traffic light 1–9

Important notices

A report is a programme, not a single value.

Before the consultation

During the consultation

Afterwards

Your four modifiable values.

All genetic evaluations

Methylation

LINE-1DNA stability

Inflammation regulation (IL-6 & TNF-α)

microRNA

miR-21Inflammation and micronutrients

miR-155FTO regulation and adipose tissue formation

Metabolic type: balanced type

Exercise type: endurance type

Weight loss through exercise

Weight loss through exerciseFTO rs1121980

Predisposition: body weight regulation and blood sugar metabolism

Body weight regulation

Blood sugar and insulin metabolism

Nutrition type

Food cravings

Hunger and satiety regulation

Losing weight

Maintaining weight

Vitamin uptake

FolateMTHFR

Vitamin DTransport

B vitaminsRequirement

Omega 3 and blood lipids

Blood lipids and fatty acid metabolism

Salt sensitivity

Salt sensitivity

Nutricosmetics

Skin hydration

Oxidative cell protection

Skin elasticity and inflammation

Antioxidant capacity

All genes analysed

Your three focus areas.

Methylation metabolism — folate, B vitamins, zinc

Fat quality — EPA and DHA directly

Blood sugar quality rather than calorie quantity

Questions about your report?